Svenja Fritzlar

Downregulation of MHC Class I Expression by Influenza A and B Viruses

Marios Koutsakos, Hamish EG McWilliam, Turgut E Aktepe, Svenja Fritzlar, Patricia T Illing, Nicole A Mifsud, Anthony W Purcell, Steve Rockman, Patrick C Reading, Julian P Vivian, Jamie Rossjohn, Andrew G Brooks, Jason M Mackenzie, Justine D Mintern, Jose A Villadangos, Thi HO Nguyen, Katherine Kedzierska

Journal article  |  2019  |  Frontiers in Immunology

Mouse Norovirus Infection Arrests Host Cell Translation Uncoupled from the Stress Granule-PKR-eIF2 alpha Axis

Svenja Fritzlar, Turgut E Aktepe, Yi-Wei Chao, Nathan D Kenney, Michael R McAllaster, Craig B Wilen, Peter A White, Jason M Mackenzie

Journal article  |  2019  |  mBio

The Microtubule-Associated Innate Immune Sensor GEF-H1 Does Not Influence Mouse Norovirus Replication in Murine Macrophages

Svenja Fritzlar, Peter A White, Jason M Mackenzie

Journal article  |  2019  |  Viruses

Mouse Norovirus Infection Reduces the Surface Expression of Major Histocompatibility Complex Class I Proteins and Inhibits CD8( ) T Cell Recognition and Activation

Svenja Fritzlar, Sinthujan Jegaskanda, Turgut Esad Aktepe, Julia Emiley Prier, Lauren Elise Holz, Peter A White, Jason M Mackenzie

Journal article  |  2018  |  Journal of Virology

Interaction of mouse norovirus (MNV) with the cellular immune response of host cells

Svenja Fritzlar

Thesis / Dissertation  |  2017

Human noroviruses (HuNoV) cause the majority of non-bacterial gastroenteritis cases worldwide and generate an economic burden of 6..

Norovirus Infection: Replication, Manipulation of Host, and Interaction with the Host Immune Response

Soroush T Sarvestani, Ben Cotton, Svenja Fritzlar, Tanya B O'Donnell, Jason M Mackenzie

Journal article  |  2016  |  Journal of Interferon & Cytokine Research

Increased Survival in the Red Flour Beetle after Oral Priming with Bacteria-Conditioned Media

Barbara Milutinovic, Svenja Fritzlar, Joachim Kurtz

Journal article  |  2014  |  Journal of Innate Immunity

Immune priming is defined as enhanced protection upon secondary exposure to a pathogen. Such enhanced resistance after prior expos..