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Email

april.watt@unimelb.edu.au

Credentials


Position
Research Fellow
The Sir Peter MacCallum Department of Oncology
Education
Doctorate
University of Melbourne
Masters (Research)
Carnegie Mellon University
Bachelors Degree
Carnegie Mellon University
ORCID

0000-0002-5145-5334

Dr April Watt

Research Fellow
The Sir Peter MacCallum Department of Oncology

11 Scholarly works
0 Projects

HIGHLIGHTS

  • 2026

    Journal article

    Abstract GS3-06: Rb Functions as a Transcriptional Activator of ER Targets Following CDK4/6 Inhibition in Luminal Breast Cancer
    DOI: 10.1158/1557-3265.sabcs25-gs3-06
  • 2025

    Journal article

    Correction: INX-315, a Selective CDK2 Inhibitor, Induces Cell Cycle Arrest and Senescence in Solid Tumors(Cancer Discov 2024, 14,446–67.)
    DOI: 10.1158/2159-8290.CD-25-1502
  • 2024

    Thesis / Dissertation

    Epigenetic Consequences of CDK4/6 Inhibition in Breast Cancer
  • 2024

    Journal article

    INX-315, a Selective CDK2 Inhibitor, Induces Cell Cycle Arrest and Senescence in Solid Tumors
    DOI: 10.1158/2159-8290.CD-23-0954
  • 2022

    Journal article

    Cellular mechanisms underlying response and resistance to CDK4/6 inhibitors in the treatment of hormone receptor-positive breast cancer
    DOI: 10.1186/s13058-022-01510-6
  • 2022

    Journal article

    Analysis of functional surfaces on the actin nucleation promoting factor Dip1 required for Arp2/3 complex activation and endocytic actin network assembly
    DOI: 10.1016/j.jbc.2022.102019
  • 2021

    Journal article

    CDK4/6 inhibition reprograms the breast cancer enhancer landscape by stimulating AP-1 transcriptional activity
    DOI: 10.1038/s43018-020-00135-y
April Watt

RECENT SCHOLARLY WORKS

  • 2018

    Conference Proceedings

    CDK4/6 inhibition triggers an antitumor immune response
    DOI: 10.1158/2326-6074.TUMIMM17-B04
  • 2017

    Journal article

    CDK4/6 inhibition triggers anti-tumour immunity
    DOI: 10.1038/nature23465
  • 2017

    Conference Proceedings

    CDK4/6 inhibition directly enhances an anti-tumor immune response in breast cancer
    DOI: 10.1158/1538-7445.AM2017-1634

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