Ms. Michele Binder
Honorary
Anatomy and Physiology
34 Scholarly works
5 Projects
HIGHLIGHTS
2021
Journal article
Neuregulin therapy for multiple sclerosis: an each-way bet? COMMENT
DOI: 10.1093/brain/awaa4342021
Journal article
miRNAs in Microglia: Important Players in Multiple Sclerosis Pathology
DOI: 10.1177/17590914209811822020
Journal article
Multiple Sclerosis as a Syndrome-Implications for Future Management
DOI: 10.3389/fneur.2020.007842020
Journal article
Tyro3 Contributes to Retinal Ganglion Cell Function, Survival and Dendritic Density in the Mouse Retina
DOI: 10.3389/fnins.2020.008402020
Research Grant
Dendritic cell therapy for multiple sclerosis
2018
Journal article
The TAM receptor TYRO3 is a critical regulator of myelin thickness in the central nervous system
DOI: 10.1002/glia.234812012
Research Grant
Molecules that regulate demyelination
RECENT SCHOLARLY WORKS
2017
Journal article
The TAM Receptor Tyro3 Regulates Myelination in the Central Nervous System
DOI: 10.1002/glia.231132016
Journal article
Common and Low Frequency Variants in MERTK Are Independently Associated with Multiple Sclerosis Susceptibility with Discordant Association Dependent upon HLA-DRB1*15:01 Status
DOI: 10.1371/journal.pgen.10058532016
Journal article
Common and Low Frequency Variants in MERTK Are Independently Associated with Multiple Sclerosis Susceptibility with Discordant Association Dependent upon HLA-DRB1*15:01 Status
DOI: 10.1371/journal.pgen.10058532015
Conference Proceedings
The TAM receptor Tyro3 is a positive regulator of myelination
2015
Conference Proceedings
Mertk deficiency in dendritic cells exacerbates experimental autoimmune encephalomyelitis
RECENT PROJECTS
2011
Research Grant
Use of gene expression profiles to characterise T lymphocyte activity in patients during their first demyelinating event and the likelihood of conversion to multiple sclerosis.
2009
Research Grant
Analysing how key molecules protect the nervous system in Multiple Sclerosis
2009
Research Grant
To determine whether changes in TAM receptor signalling genes influence susceptibility to MS