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Email

emma.carrington@unimelb.edu.au

Credentials


Position
Honorary (Fellow)
Faculty of Medicine, Dentistry and Health Sciences - Admin

Dr Emma Carrington

Honorary (Fellow)
Faculty of Medicine, Dentistry and Health Sciences

28 Scholarly works
0 Projects

HIGHLIGHTS

  • 2021

    Journal article

    BCL-XL antagonism selectively reduces neutrophil life span within inflamed tissues without causing neutropenia
    DOI: 10.1182/BLOODADVANCES.2020004139
  • 2020

    Journal article

    Adoptive cellular therapy with T cells expressing the dendritic cell growth factor Flt3L drives epitope spreading and antitumor immunity
    DOI: 10.1038/s41590-020-0676-7
  • 2019

    Journal article

    Recipient BCL2 inhibition and NK cell ablation form part of a reduced intensity conditioning regime that improves allo-bone marrow transplantation outcomes
    DOI: 10.1038/s41418-018-0228-y
  • 2018

    Journal article

    GM-CSF Quantity Has a Selective Effect on Granulocytic vs. Monocytic Myeloid Development and Function
    DOI: 10.3389/fimmu.2018.01922
  • 2017

    Journal article

    Life and death of activated T cells: How are they different from naïve T Cells?
    DOI: 10.3389/fimmu.2017.01809
  • 2017

    Journal article

    DNA-binding of the Tet-transactivator curtails antigen-induced lymphocyte activation in mice
    DOI: 10.1038/s41467-017-01022-4
  • 2017

    Journal article

    The life and death of immune cell types: The role of BCL-2 anti-apoptotic molecules
    DOI: 10.1038/icb.2017.72
Emma Carrington

RECENT SCHOLARLY WORKS

  • 2017

    Journal article

    Anti-apoptotic proteins BCL-2, MCL-1 and A1 summate collectively to maintain survival of immune cell populations both in vitro and in vivo
    DOI: 10.1038/cdd.2017.30
  • 2017

    Journal article

    Cognate antigen engagement on parenchymal cells stimulates CD8 T cell proliferation in situ
    DOI: 10.1038/ncomms14809
  • 2017

    Journal article

    Characterisation of mice lacking all functional isoforms of the pro-survival BCL-2 family member A1 reveals minor defects in the haematopoietic compartment
    DOI: 10.1038/cdd.2016.156

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