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Email

hanneke.raaijmakers@unimelb.edu.au

Credentials


Position
Research Fellow
Department of Anatomy and Physiology
ORCID

0000-0003-0827-2441

Ms Hanneke Raaijmakers

Research Fellow
Department of Anatomy and Physiology

58 Scholarly works
0 Projects

HIGHLIGHTS

  • 2026

    Journal article

    Female HFpEF diastolic dysfunction and premature mortality is accentuated relative to male in an experimental HFpEF model – a specific cardiac lipid accumulation
    DOI: 10.1016/j.jmccpl.2025.100779
  • 2026

    Journal article

    Proteomic analysis indicates capacity for epicardial adipose paracrine influence in the hearts of healthy, lean sheep
    DOI: 10.1016/j.jmccpl.2025.100790
  • 2025

    Journal article

    Targeted glycophagy ATG8 therapy reverses diabetic heart disease in mice and in human engineered cardiac tissues
    DOI: 10.1038/s44161-025-00726-x
  • 2024

    Journal article

    Mechanical loading reveals an intrinsic cardiomyocyte stiffness contribution to diastolic dysfunction in murine cardiometabolic disease
    DOI: 10.1113/JP286437
  • 2024

    Journal article

    Sex-specific regulation of the cardiac transcriptome by the protein phosphatase 2A regulatory subunit B55α
    DOI: 10.1038/s44324-024-00033-2
  • 2024

    Journal article

    Investigating STarch Binding Domain Containing Protein 1 (STBD1) as a Physiologic Regulator of Cardiac Functional and Metabolic Homeostasis
    DOI: 10.1016/j.hlc.2024.06.775
  • 2020

    Journal article

    Epicardial Adipose Tissue Accumulation Confers Atrial Conduction Abnormality
    DOI: 10.1016/j.jacc.2020.07.017
Hanneke Raaijmakers

RECENT SCHOLARLY WORKS

  • 2024

    Journal article

    Sheep Epicardial Adipose Exerts Paracrine Influence to Increase Integrin-Linked Kinase Phosphorylation in HL-1 Cells
    DOI: 10.1016/j.hlc.2024.06.783
  • 2024

    Journal article

    Glycophagy is an Effective Therapeutic Target to Remediate Diastolic Dysfunction in Diabetic Heart Disease
    DOI: 10.1016/j.hlc.2024.06.767
  • 2024

    Journal article

    The cardiomyocyte origins of diastolic dysfunction: cellular components of myocardial “stiffness”
    DOI: 10.1152/ajpheart.00334.2023
  • 2024

    Journal article

    A gene therapy targeting medium-chain acyl-CoA dehydrogenase (MCAD) did not protect against diabetes-induced cardiac pathology
    DOI: 10.1007/s00109-023-02397-2

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