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Email

helen.kiriazis@unimelb.edu.au

Credentials


Position
Honorary (Senior Fellow)
Baker Department of Cardiometabolic Health

Ms Helen Kiriazis

Honorary (Senior Fellow)
Baker Department of Cardiometabolic Health

14 Scholarly works
0 Projects

HIGHLIGHTS

  • 2025

    Journal article

    Annexin-A1 deficiency uncovers female-specific pathways in blood pressure control and cardiovascular remodeling in mice
    DOI: 10.1038/s42003-025-08291-6
  • 2025

    Journal article

    Multiparity induces persistent myocardial structural, functional and transcriptomic remodelling in mice
    DOI: 10.1038/s41598-025-08248-z
  • 2025

    Journal article

    Reduced PI3K(p110α) induces atrial myopathy, and PI3K-related lipids are dysregulated in athletes with atrial fibrillation
    DOI: 10.1016/j.jshs.2025.101023
  • 2025

    Journal article

    Acute Inhibition of Drp1 Mitigates Adverse Cardiac Remodelling following Chronic Reperfused Myocardial Infarction
    DOI: 10.1007/s10557-024-07633-6
  • 2025

    Journal article

    Generation and evaluation of a novel PI3K-targeting gene therapy in the failing mouse heart and healthy sheep heart
    DOI: 10.1016/j.jmccpl.2025.100478
  • 2025

    Journal article

    The Murine Heart Undergoes Profound Lipidomic Remodelling 24 Hours Post Ischaemia Reperfusion Injury
    DOI: 10.1016/j.hlc.2025.06.842
  • 2025

    Journal article

    Reduced PI3K and Anthracycline Treatment Prevents Exercise-Induced Cardiac Hypertrophy
    DOI: 10.1016/j.hlc.2025.06.384
Helen Kiriazis

RECENT SCHOLARLY WORKS

  • 2025

    Journal article

    An optimized plasmalogen modulating dietary supplement provides greater protection in a male than female mouse model of dilated cardiomyopathy
    DOI: 10.1016/j.jmccpl.2024.100273
  • 2024

    Journal article

    Sex-specific regulation of the cardiac transcriptome by the protein phosphatase 2A regulatory subunit B55α
    DOI: 10.1038/s44324-024-00033-2
  • 2024

    Journal article

    Distinct functional and molecular profiles between physiological and pathological atrial enlargement offer potential new therapeutic opportunities for atrial fibrillation
    DOI: 10.1042/CS20240178

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