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Email

andrea.newbold@unimelb.edu.au

Credentials


Position
Honorary (Fellow)
The Sir Peter MacCallum Department of Oncology
ORCID

0000-0002-0481-2431

Dr Andrea Newbold

Honorary (Fellow)
The Sir Peter MacCallum Department of Oncology

42 Scholarly works
0 Projects

HIGHLIGHTS

  • 2026

    Journal article

    Dual Bcl-2/Bcl-xl inhibition via AZD0466 combines with immune checkpoint blockade to enhance anti-tumour activity
    DOI: 10.1038/s41419-025-08354-w
  • 2025

    Journal article

    A CDK11-dependent RNA polymerase II pause-checkpoint precedes CDK9-mediated transition to transcriptional elongation
    DOI: 10.1016/j.molcel.2025.08.001
  • 2023

    Conference Proceedings

    P1212: RATIONALLY DESIGNED CHIMERIC PI3K-BET BROMODOMAIN INHIBITORS ELICIT CURATIVE RESPONSES IN MYC-DRIVEN LYMPHOMA
    DOI: 10.1097/01.hs9.0000971744.43694.69
  • 2023

    Journal article

    Rationally designed chimeric PI3K-BET bromodomain inhibitors elicit curative responses in MYC-driven lymphoma
    DOI: 10.1073/pnas.2306414120
  • 2023

    Journal article

    Stepwise progression of β-selection during T cell development involves histone deacetylation
    DOI: 10.26508/lsa.202201645
  • 2022

    Journal article

    The Combination of Curaxin CBL0137 and Histone Deacetylase Inhibitor Panobinostat Delays KMT2A-Rearranged Leukemia Progression
    DOI: 10.3389/fonc.2022.863329
  • 2021

    Journal article

    The PP2A-Integrator-CDK9 axis fine-tunes transcription and can be targeted therapeutically in cancer
    DOI: 10.1016/j.cell.2021.04.022
Andrea Newbold

RECENT SCHOLARLY WORKS

  • 2021

    Journal article

    Serine biosynthesis is a metabolic vulnerability in flt3-itd–driven acute myeloid leukemia
    DOI: 10.1158/2159-8290.CD-20-0738
  • 2020

    Journal article

    AZD4320, a dual inhibitor of bcl-2 and bcl-xl, induces tumor regression in hematologic cancer models without dose-limiting thrombocytopenia
    DOI: 10.1158/1078-0432.CCR-20-0863
  • 2020

    Conference Proceedings

    REPROGRAMMING OF SERINE METABOLISM IS A METABOLIC VULNERABILITY IN FMS-LIKE TYROSINE KINASE 3 (FLT3) MUTANT ACUTE MYELOID LEUKAEMIA
    DOI: 10.1016/j.exphem.2020.09.024

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