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Email

holly.barker@unimelb.edu.au

Credentials


Position
Honorary (Fellow)
Department of Medical Biology (WEHI)

Dr Holly Barker

Honorary (Fellow)
Department of Medical Biology (WEHI)

42 Scholarly works
0 Projects

HIGHLIGHTS

  • 2026

    Journal article

    High-throughput drug screening identifies EGFR/MAPK pathway targeting sensitivities in organoid models of ovarian carcinosarcoma
    DOI: 10.1186/s13046-025-03629-8
  • 2025

    Journal article

    Genomics of cervical, vulvar and vaginal cancers and the potential of precision medicine
    DOI: 10.1177/17588359251363499
  • 2025

    Journal article

    Genomics of ovarian cancers and the potential of precision medicine
    DOI: 10.1177/17588359251396651
  • 2025

    Journal article

    Genomics of uterine malignancies and the potential of precision medicine
    DOI: 10.1177/17588359251387395
  • 2024

    Journal article

    BRCA1 secondary splice-site mutations drive exon-skipping and PARP inhibitor resistance
    DOI: 10.1186/s12943-024-02048-1
  • 2024

    Journal article

    CHK1 inhibitor SRA737 is active in PARP inhibitor resistant and CCNE1 amplified ovarian cancer
    DOI: 10.1016/j.isci.2024.109978
  • 2017

    Journal article

    Secondary somatic mutations restoring RAD51C and RAD51D associated with acquired resistance to the PARP inhibitor rucaparib in high-grade ovarian carcinoma
    DOI: 10.1158/2159-8290.CD-17-0419
Holly Barker

RECENT SCHOLARLY WORKS

  • 2024

    Journal article

    A risk-reward examination of sample multiplexing reagents for single cell RNA-Seq
    DOI: 10.1016/j.ygeno.2024.110793
  • 2023

    Journal article

    Targeting homologous recombination deficiency in uterine leiomyosarcoma
    DOI: 10.1186/s13046-023-02687-0
  • 2023

    Journal article

    A Matched Molecular and Clinical Analysis of the Epithelioid Haemangioendothelioma Cohort in the Stafford Fox Rare Cancer Program and Contextual Literature Review
    DOI: 10.3390/cancers15174378
  • 2023

    Journal article

    SF3B1 hotspot mutations confer sensitivity to PARP inhibition by eliciting a defective replication stress response
    DOI: 10.1038/s41588-023-01460-5

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