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Email

monique.smeets@unimelb.edu.au

Credentials


Position
Honorary (Fellow)
Department of Medicine
Education
PhD
Free University of Amsterdam
ORCID

0000-0001-6027-4108

Miss Monique Smeets

Honorary (Fellow)
Department of Medicine

41 Scholarly works
0 Projects

HIGHLIGHTS

  • 2026

    Journal article

    KLHDC3 deficiency in mice reveals essential roles in development, survival, and adiposity via the DesCEND ubiquitin pathway
    DOI: 10.1186/s12864-026-12574-5
  • 2026

    Journal article

    Minute amounts of helicase-deficient truncated RECQL4 are sufficient for DNA replication
    DOI: 10.1038/s44319-026-00727-2
  • 2025

    Journal article

    Characteristics of the Kelch domain containing (KLHDC) subfamily and relationships with diseases
    DOI: 10.1002/1873-3468.15108
  • 2024

    Journal article

    3193 – UNDERSTANDING HOW LOSS OF RECQL4 IS SUPPRESSED BY LOSS OF THE CRL2 SUBSTRATE RECEPTOR KLHDC3
    DOI: 10.1016/j.exphem.2024.104513
  • 2024

    Journal article

    3192 – ADAR1P150 PROTEIN, INDEPENDENT OF ITS A-TO-I RNA EDITING ACTIVITY, REGULATES PERIPHERAL T CELL HOMEOSTASIS AND HSC REPOPULATING CAPACITY
    DOI: 10.1016/j.exphem.2024.104512
  • 2022

    Journal article

    Srsf2P95H/ co-operates with loss of TET2 to promote myeloid bias and initiate a chronic myelomonocytic leukemia-like disease in mice
    DOI: 10.1038/s41375-022-01727-6
  • 2022

    Journal article

    Genome-wide screening identifies cell-cycle control as a synthetic lethal pathway with SRSF2P95H mutation
    DOI: 10.1182/bloodadvances.2021004571
Monique Smeets

RECENT SCHOLARLY WORKS

  • 2020

    Journal article

    3148 – SRSF2P95H AND TET2 LOSS CO-OPERATE TO PROMOTE CHRONIC MYELOMONOCYTIC LEUKAEMIA IN VIVO
    DOI: 10.1016/j.exphem.2020.09.155
  • 2020

    Journal article

    3059 – PROTEIN TRUNCATING, BUT NOT HELICASE INACTIVATING, RECQL4 MUTATIONS CAUSE BONE MARROW FAILURE
    DOI: 10.1016/j.exphem.2020.09.076
  • 2020

    Conference Proceedings

    DNA REPAIR AND CELL CYCLE ARE SYNTHETIC LETHAL PATHWAYS IN SRSF2P95H MUTATED CELLS
    DOI: 10.1016/j.exphem.2020.09.156

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