No pain no word gain: toward a newÂ neurobiological account of word learning
Grant number: DE180100893 | Funding period: 2018 - 2021
This project aims to generate a novel neurobiological account of word learning, going beyond a simple mapping between words and objects and recognising the sensory and socio-communicative embedding of language. Capitalising on interdisciplinary approaches to research, this project will use state-of-the-art neuroimaging to reveal the neural architecture and mechanisms supporting contextualised sensory word learning. The results are expected to bring about a paradigm shift in the fields of neurobiology of language and learning, having a profound impact on the practice of language teaching and improvement of language functioning.
Related publications (14)
Regional neurodegeneration correlates with sleep-wake dysfunction after stroke.
Elie Gottlieb, Natalia Egorova, Mohamed S Khlif, Wasim Khan, Emilio Werden, Matthew P Pase, Mark Howard, Amy Brodtmann
Sleep-wake disruption is a key modifiable risk factor and sequela of stroke. The pathogenesis of poststroke sleep dysfunction is u..
Dynamic Regional Brain Atrophy Rates in the First Year After Ischemic Stroke
Amy Brodtmann, Mohamed Salah Khlif, Natalia Egorova, Michele Veldsman, Laura J Bird, Emilio Werden
BACKGROUND AND PURPOSE: Brain atrophy can be regarded as an end-organ effect of cumulative cardiovascular risk factors. Accelerate..
Three-tissue compositional analysis reveals in-vivo microstructural heterogeneity of white matter hyperintensities following stroke
Wasim Khan, Natalia Egorova, Mohamed Salah Khlif, Remika Mito, Thijs Dhollander, Amy Brodtmann
White matter hyperintensities (WMHs) are frequently observed on brain scans of older individuals and are associated with cognitive..
Pervasive White Matter Fiber Degeneration in Ischemic Stroke
Natalia Egorova, Thijs Dhollander, Mohamed Salah Khlif, Wasim Khan, Emilio Werden, Amy Brodtmann
Background and Purpose- We examined if ischemic stroke is associated with white matter degeneration predominantly confined to the ..
Microstructural degeneration underlies executive dysfunction after stroke
Michele Veldsman, Emilio Werden, Natalia Egorova, Mohamed Salah Khlif, Amy Brodtmann
<jats:title>ABSTRACT</jats:title><jats:sec><jats:title>Objective</jats:title><jats:p>Executive dysfunction affects 40% of stroke p..
A Human Depression Circuit Derived From Focal Brain Lesions
Jaya L Padmanabhan, Danielle Cooke, Juho Joutsa, Shan H Siddiqi, Michael Ferguson, R Ryan Darby, Louis Soussand, Andreas Horn, Na Young Kim, Joel L Voss, Andrew M Naidech, Amy Brodtmann, Natalia Egorova, Sophia Gozzi, Thanh G Phan, Maurizio Corbetta, Jordan Grafman, Michael D Fox
BACKGROUND: Focal brain lesions can lend insight into the causal neuroanatomical substrate of depression in the human brain. Howev..
A comparison of automated segmentation and manual tracing in estimating hippocampal volume in ischemic stroke and healthy control participants
Mohamed Salah Khlif, Natalia Egorova, Emilio Werden, Alberto Redolfi, Marina Boccardi, Charles S DeCarli, Evan Fletcher, Baljeet Singh, Qi Li, Laura Bird, Amy Brodtmann
APOE epsilon 4 Carriers Show Delayed Recovery of Verbal Memory and Smaller Entorhinal Volume in the First Year After Ischemic Stroke
Emilio Werden, Mohamed Salah Khlif, Laura J Bird, Toby Cumming, Jennifer Bradshaw, Wasim Khan, Matthew Pase, Carolina Restrepo, Michele Veldsman, Natalia Egorova, Sheila K Patel, Elie Gottlieb, Amy Brodtmann
Assessment of longitudinal hippocampal atrophy in the first year after ischemic stroke using automatic segmentation techniques
Mohamed Salah Khlif, Emilio Werden, Natalia Egorova, Marina Boccardi, Alberto Redolfi, Laura Bird, Amy Brodtmann
We assessed first-year hippocampal atrophy in stroke patients and healthy controls using manual and automated segmentations: AdaBo..