Journal article

ATF-2 IS PREFERENTIALLY ACTIVATED BY STRESS-ACTIVATED PROTEIN-KINASES TO MEDIATE C-JUN INDUCTION IN RESPONSE TO GENOTOXIC AGENTS

H VANDAM, D WILHELM, I HERR, A STEFFEN, P HERRLICH, P ANGEL

The EMBO Journal | WILEY | Published : 1995

DOI: 10.1002/j.1460-2075.1995.tb07168.x

University of Melbourne Researchers

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Keywords

Transcription, Genetic
Mitogen-Activated Protein Kinase Kinases
E1a Proteins
Glucocorticoid Receptor
Stress-Activated Protein Kinases
Fos Jun
Map Kinase Kinase 4
Molecular Sequence Data
Transcriptional Activation
Atf-2
Methyl Methanesulfonate
Transcription Factor Family
Cyclic Amp Response Element-Binding Protein
Gene Expression Regulation
Gene-Transcription
Methylnitronitrosoguanidine
Activating Transcription Factor 2
Protein Kinases
Life Sciences & Biomedicine
Models, Genetic
Transcription Factors
Base Sequence
Heterodimer Formation
Threonine
Cells, Cultured
Genotoxic Stress
Complex-Formation
Cell Biology
Humans
Nf-Kappa-B
Signal Transduction
Dna Damage
Ultraviolet Rays
Transcription
Structure-Activity Relationship
Biochemistry & Molecular Biology
Cell Differentiation
C-Jun
Functional Antagonism
Dna-Binding
Phorbol Esters
Science & Technology
Proto-Oncogene Proteins C-Jun
Phosphorylation