Anti-apoptotic proteins BCL-2, MCL-1 and A1 summate collectively to maintain survival of immune cell populations both in vitro and in vivo
Emma M Carrington, Yifan Zhan, Jamie L Brady, Jian-Guo Zhang, Robyn M Sutherland, Natasha S Anstee, Robyn L Schenk, Ingela B Vikstrom, Rebecca B Delconte, David Segal, Nicholas D Huntington, Philippe Bouillet, David M Tarlinton, David CS Huang, Andreas Strasser, Suzanne Cory, Marco J Herold, Andrew M Lew
Cell Death & Differentiation | NATURE PUBLISHING GROUP | Published : 2017
Awarded by National Health and Medical Research Council of Australia (NHMRC)
Awarded by Leukemia and Lymphoma Society (LLS) Specialized Center of Research grant
Awarded by Cancer Council Victoria (CCV)
Awarded by NHMRC Independent Research Institutes Infrastructure Support Scheme grant
We thank M Dayton, M Iliopoulos, T Mason, H Johnson and M Hancock for technical assistance. This work was supported by Rebecca L. Cooper Foundation, National Health and Medical Research Council of Australia (NHMRC) grants and fellowships #1037321, #1043414, #1080321 and #1105209 to AML, #461221 and #1042629 to PB, #1020363 and #1016701 to AS, #1054925 and #1060675 to DT, #1016647 to JGZ, #1049720 to MH, the Leukemia and Lymphoma Society (LLS) Specialized Center of Research grant (#7001-13 to AS), Cancer Council Victoria (CCV) Grant-in-aid (#1102104 to AS), Austrian Academy of Science (OAW) Doc-fellowship (to MH), Leukemia Foundation National Research Program PhD Scholarship (to RS), NHMRC Independent Research Institutes Infrastructure Support Scheme grant (361646) and Victorian State Government Operational Infrastructure Support grant. We acknowledge the Wurundjeri people of the Kulin nation as the traditional owners and custodians of the land on which most of the work was performed.