Journal article

The accumulation of enzymatically inactive cuproenzymes is a CNS-specific phenomenon of the SOD1(G37R) mouse model of ALS and can be restored by overexpressing the human copper transporter hCTR1

James B Hilton, Kai Kysenius, Anthony R White, Peter J Crouch

Experimental Neurology | ACADEMIC PRESS INC ELSEVIER SCIENCE | Published : 2018