Journal article

Disabling an integral CTL epitope allows suppression of autoimmune diabetes by intranasal proinsulin peptide

NR Martinez, P Augstein, AK Moustakas, GK Papadopoulos, S Gregori, L Adorini, DC Jackson, LC Harrison

JOURNAL OF CLINICAL INVESTIGATION | AMER SOC CLINICAL INVESTIGATION INC | Published : 2003

DOI: 10.1172/JCI200317166

University of Melbourne Researchers

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Keywords

Epitopes
Myelin Basic-Protein
Diabetes Mellitus, Type 1
Peptides
Protein Binding
Adoptive Transfer
Animals
Models, Molecular
Disease
Mice
Research & Experimental Medicine
Autoantigen
Female
Life Sciences & Biomedicine
Protein Structure, Tertiary
Molecule
Mice, Inbred Nod
Beta-Cell Function
Immunology Not Elsewhere Classified; Infectious Diseases
Mhc Class-I
Administration, Intranasal
Tolerance
Humans
Cells, Cultured
Proinsulin
Oral Insulin
Nod Mice
Science & Technology
T-Lymphocytes, Cytotoxic
T-Cells
Medicine, Research & Experimental
Spleen
Male