Journal article

Transient neuroprotection by minocycline following traumatic brain injury is associated with attenuated microglial activation but no changes in cell apoptosis or neutrophil infiltration

Nicole Bye, Mark D Habgood, Jennifer K Callaway, Nakisa Malakooti, Ann Potter, Thomas Kossmann, M Cristina Morganti-Kossmann

Experimental Neurology | ACADEMIC PRESS INC ELSEVIER SCIENCE | Published : 2007

DOI: 10.1016/j.expneurol.2006.10.013

University of Melbourne Researchers

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157Web of Science
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Keywords

Proinflammatory Cytokine Expression
Motor Activity
Animals
Mice, Inbred C57bl
Neutrophil Infiltration
Head Injuries, Closed
Neurosciences
Closed-Head Injury
Life Sciences & Biomedicine
Traumatic Brain Injury
Neurosciences & Neurology
Brain Injuries
Apoptosis
Cell Count
Cytokines
Cytochrome-C Release
Improves Functional Recovery
Science & Technology
Cerebral Cortex
Interleukin-1 Receptor Antagonist
Focal Cerebral-Ischemia
Interleukin-1beta
Microglia
Neuroprotective Agents
Time Factors
Neutrophils
Mice
Minocycline
Tnf-Alpha
Nervous System
Mouse Model
Death
Brain
Male
Macrophages
Inflammation
Spinal-Cord-Injury