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Journal article

Selective intracellular release of copper and zinc ions from bis( thiosemicarbazonato) complexes reduces levels of Alzheimer disease amyloid-beta peptide

Paul S Donnelly, Aphrodite Caragounis, Tai Du, Katrina M Laughton, Irene Volitakis, Robert A Cherny, Robyn A Sharples, Andrew F Hill, Qiao-Xin Li, Colin L Masters, Kevin J Barnham, Anthony R White

Journal of Biological Chemistry | AMER SOC BIOCHEMISTRY MOLECULAR BIOLOGY INC | Published : 2008

DOI: 10.1074/jbc.M705957200

University of Melbourne Researchers

Qiao-Xin Li's Profile Picture
Qiao-Xin Li Author Melbourne Medical School

Kevin Barnham's Profile Picture
Kevin Barnham Author Florey Department of Neuroscience and Mental Health

Colin Masters's Profile Picture
Colin Masters Author Florey Department of Neuroscience and Mental Health

Paul Donnelly's Profile Picture
Paul Donnelly Author Chemistry

Robert Cherny's Profile Picture
Robert Cherny Author Clinical Pathology

Citation metrics

147Web of Science
148Scopus
135Dimensions

Keywords

Copper
Dose-Response Relationship, Drug
Alzheimer Disease
Biological Availability
Cho Cells
Thiosemicarbazones
Oxidation-Reduction
Humans
Cells
Science & Technology
Agents
Transgenic Mice
Neurodegeneration
Amyloid Beta-Protein Precursor
Zinc
Accumulation
Cricetinae
Bis(thiosemicarbazone) Complexes
Gene Expression
Binding
Drug Evaluation, Preclinical
Life Sciences & Biomedicine
Cricetulus
Brain
Metal-Complexes
Biochemistry & Molecular Biology
Cu(ii)
Animals

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