Glycosylation of the hemagglutinin modulates the sensitivity of H3N2 influenza viruses to innate proteins in airway secretions and virulence in mice

Grants

Funding Acknowledgements

This study was supported by Project Grant 4509230 from the National Health and Medical Research Council (NHMRC) of Australia. PCR is a NHMRC R.D Wright Research Fellow. The Melbourne WHO Collaborating Centre for Reference and Research on Influenza is supported by the Australian Government Department of Health and Ageing. The authors wish to thank Dr. Robert Webster, St Jude Children's Research Hospital, Memphis, TN, USA for provision of the plasmid vector used to create the reverse engineered viruses for this study.