Cytosolic caspases mediate mislocalised SOD2 depletion in an in vitro model of chronic prion infection
Layla Sinclair, Victoria Lewis, Steven J Collins, Cathryn L Haigh
DISEASE MODELS & MECHANISMS | COMPANY OF BIOLOGISTS LTD | Published : 2013
Related Projects (7)
DETERMINING NORMAL PRION PROTEIN FUNCTION, EMPHASISING THE ROLE PLAYED BY PROTEIN CLEAVAGE, AND ALSO DEFINING THE RELATIONSHIP BETWEEN SURVEILLANCE INTENSITY AND ANNUAL SPORADIC CJD INCIDENCE
As a neurologist undertaking research into prion diseases over an extended period, I have been able to lead and participate in many projects..
Awarded by National Health and Medical Research Council (NHMRC)
This work was funded by a National Health and Medical Research Council (NH&MRC) program grant (#628946) and a Brain Foundation research grant. S.J.C. is funded by an NH&MRC Practitioner Fellowship (#APP1005816). L.S. is funded in part by a Carol Willesee Foundation Ph.D. scholarship. V.L. is supported by an NH&MRC Training Fellowship (#567123).