Journal article

Loss of pro-apoptotic Bim promotes accumulation of pulmonary T lymphocytes and enhances allergen-induced goblet cell metaplasia

Joseph Pierce, Jules Rir-Sima-Ah, Isaac Estrada, Julie Wilder, Andreas Strasser, Yohannes Tesfaigzi

Am. J. Physiol. Lung Cell. Mol. Physiol. | AMER PHYSIOLOGICAL SOC | Published : 2006

DOI: 10.1152/ajplung.00516.2005

University of Melbourne Researchers

Grants

Awarded by NHLBI NIH HHS

Awarded by NIEHS NIH HHS

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Keywords

Lung
Physiology
Bh3-Only Protein Bim
Science & Technology
Apoptosis Regulatory Proteins
Allergens
Mice
Brown-Norway Rats
Cd8-Positive T-Lymphocytes
Interferon-Gamma
Asthma
Family-Member Bim
Bcl-2-Like Protein 11
Th2 Cells
Metaplasia
Proapoptotic Bcl-2 Family
Animals
Transcriptional Regulation
Interferon-Gamma and Tolerance
Mucous Cell Metaplasia
Clara Cells
Male
Induced Airway Hyperreactivity
Il-13r Alpha 2
Membrane Proteins
Interleukin-13 Receptor Alpha2 Subunit
Mice, Inbred C57bl
Ovalbumin
Proto-Oncogene Proteins
Th1 Cells
Ifn-Gamma
Respiratory System
Mice, Mutant Strains
Apoptosis
Receptors, Antigen, T-Cell, Gamma-Delta
Goblet Cells
Molecular-Cloning
Murine Asthma Model
Life Sciences & Biomedicine