Journal article

Acetylcholinesterase is increased in the brains of transgenic mice expressing the C-terminal fragment (CT100) of the beta-amyloid protein precursor of Alzheimer's disease

G Sberna, J Saez-Valero, QX Li, C Czech, K Beyreuther, CL Masters, CA McLean, DH Small

Journal of Neurochemistry | WILEY | Published : 1998

DOI: 10.1046/j.1471-4159.1998.71020723.x

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Keywords

Isoenzymes
Amyloid Beta-Protein Precursor
Science & Technology
Peptides
Biochemistry & Molecular Biology
Neurons
Alzheimer Disease
Agglutinin
Life Sciences & Biomedicine
Middle Aged
Neurosciences & Neurology
Mice, Transgenic
Peptide Fragments
Neurosciences
Animals
Selective Loss
Substrate Specificity
Mice
Molecular-Forms
Senile Dementia
Choline O-Acetyltransferase
Cerebral Cortex
Alzheimer'S Disease
Amyloid Beta Protein
Downs-Syndrome
Cholinesterases
Plaques
Butyrylcholinesterase
Down Syndrome
Humans
Aged
Acetylcholinesterase
Glycosylation
Gene Expression
Aged, 80 and Over